L-Tryptophan uptake was assayed under conditions in which the aroT gene had been inactivated by deletion and the product of the aroP permease was competitively inhibition. A mutant carrying a deletion from bgl through tnaA showed negligible L-tryptophan uptake, in contrast to a strain possessing an intact tna region or to strains carrying point mutations in tna. The ability to take up L-tryptophan was not restored by lysogenizing the tna-deleted strain with lambda tna+.
Research Article| May 15 1982
Location of the gene for the low-affinity tryptophan-specific permease of Escherichia coli
Biochem J (1982) 204 (2): 617–619.
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R M Edwards, M D Yudkin; Location of the gene for the low-affinity tryptophan-specific permease of Escherichia coli. Biochem J 15 May 1982; 204 (2): 617–619. doi: https://doi.org/10.1042/bj2040617
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