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Michael W. Stoner
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Articles

Validation of GCN5L1/BLOC1S1/BLOS1 antibodies using knockout cells and tissue

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Paramesha Bugga, Michael W. Stoner, Janet R. Manning, Bellina A.S. Mushala, Nisha Bhattarai, Maryam Sharifi-Sanjani, Bradley R. Webster, Dharendra Thapa, Iain Scott
Journal: Biochemical Journal
Biochem J (2024) 481 (10): 643–651.
https://doi.org/10.1042/BCJ20230302
Published: 10 May 2024
View articletitled, Validation of GCN5L1/BLOC1S1/BLOS1 antibodies using knockout cells and tissue
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Includes: Supplementary data
Articles

Cardiomyocyte-specific deletion of GCN5L1 reduces lysine acetylation and attenuates diastolic dysfunction in aged mice by improving cardiac fatty acid oxidation

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Jackson E. Stewart, Jenna M. Crawford, William E. Mullen, Angelica Jacques, Michael W. Stoner, Iain Scott, Dharendra Thapa
Journal: Biochemical Journal
Biochem J (2024) 481 (6): 423–436.
https://doi.org/10.1042/BCJ20230421
Published: 11 March 2024
View articletitled, Cardiomyocyte-specific deletion of GCN5L1 reduces lysine acetylation and attenuates diastolic dysfunction in aged mice by improving cardiac fatty acid oxidation
Open the PDF for Cardiomyocyte-specific deletion of GCN5L1 reduces lysine acetylation and attenuates diastolic dysfunction in aged mice by improving cardiac fatty acid oxidation in another window
Articles

Loss of GCN5L1 in cardiac cells disrupts glucose metabolism and promotes cell death via reduced Akt/mTORC2 signaling

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Janet R. Manning, Dharendra Thapa, Manling Zhang, Michael W. Stoner, Javier Traba, Catherine Corey, Sruti Shiva, Michael N. Sack, Iain Scott
Journal: Biochemical Journal
Biochem J (2019) 476 (12): 1713–1724.
https://doi.org/10.1042/BCJ20190302
Published: 19 June 2019
View articletitled, Loss of GCN5L1 in cardiac cells disrupts glucose metabolism and promotes cell death via reduced Akt/mTORC2 signaling
Open the PDF for Loss of GCN5L1 in cardiac cells disrupts glucose metabolism and promotes cell death via reduced Akt/mTORC2 signaling in another window
Includes: Supplementary data
Articles

α-Lipoic acid promotes α-tubulin hyperacetylation and blocks the turnover of mitochondria through mitophagy

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Michael W. Stoner, Dharendra Thapa, Manling Zhang, Gregory A. Gibson, Michael J. Calderon, Claudette M. St. Croix, Iain Scott
Journal: Biochemical Journal
Biochem J (2016) 473 (12): 1821–1830.
https://doi.org/10.1042/BCJ20160281
Published: 10 June 2016
View articletitled, α-Lipoic acid promotes α-tubulin hyperacetylation and blocks the turnover of mitochondria through mitophagy
Open the PDF for α-Lipoic acid promotes α-tubulin hyperacetylation and blocks the turnover of mitochondria through mitophagy in another window