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Keywords: c-Fos
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Articles
Biochem J (2020) 477 (23): 4675–4688.
Published: 11 December 2020
... over the last 20 years, the development of new therapeutic strategies for the treatment of these tumors is peremptory. The over-expression of the proto-oncogene c-Fos has been observed in several CNS tumors including glioblastoma multiforme and is usually associated with a poor prognosis. Besides its...
Includes: Supplementary data
Articles
Biochem J (2015) 467 (1): 77–90.
Published: 20 March 2015
..., ANP32B ) and pharmacological inhibition of PP2A stabilized a fos reporter mRNA. Our results indicate that p38 MAPK regulates fos mRNA decay by affecting the state of phosphorylation of HuR while controlling yet to be fully elucidated PP regulatory networks. stability of mRNAs c-fos p38...
Includes: Supplementary data
Articles
Biochem J (2004) 382 (2): 545–556.
Published: 24 August 2004
..., defective in SHP-2 association, prevents ERK (extracellular-signal-regulated kinase) activation and expression of a luciferase reporter plasmid driven by the c- fos SRE (serum response element), indicating that interaction of SHP-2 with Gab2 is required for ERK activation in response to IL-2. Further...
Articles
Biochem J (2001) 360 (3): 599–607.
Published: 10 December 2001
... determine the identities of individual B-ZIP proteins in various AP-1 complexes we tested the effect of dominant-negative mutants to the B-ZIP proteins c-Fos, ATF2, ATF4 and CCAAT-enhancer-binding protein (C/EBP) on the activities of the collagenase and c-Jun promoters. These dominant-negative mutants...
Articles
Biochem J (1999) 342 (2): 275–280.
Published: 24 August 1999
... photon-counting imaging of insulin- and c- fos -promoter-firefly luciferase reporter construct activity. Normalized to constitutive viral promoter activity, insulin promoter activity in MIN6 β-cells was increased 1.6-fold after incubation at 30 mM compared with 3 mM glucose, but was unaltered at either...
Articles
Biochem J (1999) 339 (1): 167–175.
Published: 25 March 1999
... fibroblast and HaCaT c- fos and c- jun gene expression after exposure to EGF and PMA showed that although both agonists increased the expression of c- fos and c- jun mRNA in fibroblasts, only EGF did so in HaCaT keratinocytes. Finally, we looked at the activation of mitogen-activated protein (MAP) family...
Articles
Biochem J (1999) 338 (2): 427–432.
Published: 22 February 1999
...Hung-Yun LIN; Ai SHIH; Faith B. DAVIS; Paul J. DAVIS We have examined the effects of l -thyroxine (T 4 ) on the activation of signal transducer and activator of transcription 3 (STAT3) and on the STAT3-dependent induction of c-Fos expression by epidermal growth factor (EGF). T 4 , at a...