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Keywords: heart disease
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Biochem Soc Trans (2022) 50 (5): 1247–1255.
Published: 25 October 2022
... governing fibroblast activation. A better understanding of the molecular mechanisms that trigger and sustain the fibrotic process in heart disease has the potential to accelerate the development of therapies that specifically target the cardiac activated fibroblasts, which are at the moment unavailable...
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Biochem Soc Trans (2020) 48 (1): 165–177.
Published: 03 February 2020
... The Author(s). Published by Portland Press Limited on behalf of the Biochemical Society 2020 caveolae caveolin heart disease metabolism mitochondria Mitochondria are classically known as the ‘powerhouse’ of the cell, sustaining high energy needs through oxidative phosphorylation...
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Biochem Soc Trans (2007) 35 (5): 957–961.
Published: 25 October 2007
... from states of rest to activity. It is therefore not surprising that any factors that contribute to a loss of rhythm or contractility give rise to pathological consequences. Indeed, heart disease is a significant cause of mortality, being responsible, in 2004, for 137700 deaths in the U.K., equating...
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Biochem Soc Trans (2004) 32 (2): 383–386.
Published: 01 April 2004
... (GPCR) heart disease hypertrophy phosphoinositide 3-kinase γ (PI3Kγ) Abbreviations used: β-AR, β-adrenergic receptor; GPCR, G-protein-coupled receptor; PAF, platelet-activating factor; PI3K, phosphoinositide 3-kinase; PKB, protein kinase B; PTEN, phosphatase and tensin homologue deleted...
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