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Keywords: angiotensin II
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Articles
Biosci Rep (2021) 41 (12): BSR20211201.
Published: 07 December 2021
...Flavia L. Martins; Caio A.M. Tavares; Pamella A. Malagrino; Thiago Rentz; Acaris Benetti; Thiago M.S. Rios; Gabriel M.D. Pereira; Bruno Caramelli; Samantha K. Teixeira; José E. Krieger; Adriana C.C. Girardi The angiotensin-converting enzyme (ACE)/Angiotensin II (Ang II) and angiotensin-converting...
Includes: Supplementary data
Articles
Biosci Rep (2020) 40 (10): BSR20202388.
Published: 13 October 2020
... Press Limited on behalf of the Biochemical Society and distributed under the Creative Commons Attribution License 4.0 (CC BY) . Angiotensin II lymphoma phosphatidylinositol 3-kinase proliferation protein kinase B Arising from extranodal lymphoid tissue or lymph glands, malignant...
Articles
Biosci Rep (2019) 39 (5): BSR20182055.
Published: 03 May 2019
... device-based approaches . Eur. Heart J. 32 , 2739 – 2747 10.1093/eurheartj/ehr253 21951628 2. Simko F. , Simko J. and Fabryova M. ( 2003 ) ACE-inhibition and angiotensin II receptor blockers in chronic heart failure: pathophysiological consideration of the unresolved...
Includes: Supplementary data
Articles
Biosci Rep (2017) 37 (3): BSR20160624.
Published: 21 June 2017
... vascular functions and related ion channels were assessed in male offspring at 5 months old. Compared with the control, angiotensin II (AII)-induced vasoconstrictions were significantly higher in the MCA of the offspring exposed to prenatal HS. In the MCA, large conductance Ca 2+ -activated K + channels...
Articles
Biosci Rep (2015) 35 (5): e00252.
Published: 29 September 2015
...Qingbin Zhao; Junfang Zhang; Huifang Wang AngII (angiotensin II)-induced excessive ROS (reactive oxygen species) generation and proliferation of VSMCs (vascular smooth muscle cells) is a critical contributor to the pathogenesis of atherosclerosis. PGC-1α [PPARγ (peroxisome-proliferator-activated...
Includes: Supplementary data
Articles
Biosci Rep (2014) 34 (3): e00107.
Published: 09 May 2014
... and total NCC in WNK4 +/+ mice via AngII (angiotensin II) and aldosterone suppression. This was not ameliorated by WNK4 knock out, excluding the negative regulation of WNK4 on NCC postulated to be active in the absence of AngII stimulation. Thus, WNK4 is the major positive regulator of NCC in the kidneys...
Includes: Supplementary data