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Keywords: acute myocardial infarction
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Clin Sci (Lond) (2020) 134 (2): 273–287.
Published: 29 January 2020
... cardiovascular events (MACEs) by 30%. However, many residual risks still remain. To clarify the mechanism involved, we studied patients with acute myocardial infarction (AMI) with low LDL-C levels. Lymphocytes were isolated, and it was found that despite no difference in plasma LDL-C level, the lymphocyte...
Includes: Supplementary data
Clin Sci (Lond) (2019) 133 (3): 497–513.
Published: 12 February 2019
... cardiac function. These findings indicate that it could be a promising intervention with the potential to afford cardioprotection in the clinical setting of acute myocardial infarction. Correspondence: Nipon Chattipakorn ( nchattip@gmail.com ) 06 01 2019 30 01 2019 30 01 2019 31...
Clin Sci (Lond) (2015) 129 (1): 27–38.
Published: 01 April 2015
... mechanism and causal risk factors for plaque rupture remain unclear. To identify a potential molecule that can influence atherosclerotic plaque rupture, we investigated protein expression in serum from patients with acute myocardial infarction (AMI) and stable angina (SA), using proteomic analysis...
Includes: Supplementary data
Clin Sci (Lond) (2015) 128 (11): 751–760.
Published: 17 March 2015
...Qiu-Yue Han; Hong-Xia Wang; Xiao-Hong Liu; Cai-Xia Guo; Qi Hua; Xiao-Hong Yu; Nan Li; Yan-Zong Yang; Jie Du; Yun-Long Xia; Hui-Hua Li Because of the delayed release of troponins, early diagnosis of acute myocardial infarction (AMI) is a problem. E3 ligase Rnf207 showed higher sensitivity...
Includes: Supplementary data
Clin Sci (Lond) (2012) 123 (6): 387–398.
Published: 29 May 2012
.... They invade peripheral tissues to detect harmful antigens, inducing a local immune response. Studies suggest that DCPs (dendritic cell precursors) might be reduced in AMI (acute myocardial infarction); however, the reason for their reduction is unknown yet. In the present study, circulating mDCPs (myeloid...
Clin Sci (Lond) (2012) 123 (2): 111–119.
Published: 27 March 2012
...Klaus Distelmaier; Christopher Adlbrecht; Johannes Jakowitsch; Oswald Wagner; Christopher Gerner; Irene M. Lang; Markus Kubicek Thrombotic occlusion of an epicardial coronary artery on the grounds of atherosclerotic plaque is considered the ultimate step in AMI (acute myocardial infarction...
Includes: Supplementary data
Clin Sci (Lond) (2010) 119 (2): 87–95.
Published: 20 April 2010
... stable due to binding with other materials. The aim of the present translational study is to establish a method of determining the absolute amount of an miRNA in blood and to determine the potential applications of circulating cell-free miR-1 (microRNA-1) in AMI (acute myocardial infarction). The results...
Clin Sci (Lond) (2007) 113 (7): 319–327.
Published: 03 September 2007
... this effect of PTHrP-(1–36). Moreover, 10 −7 mol/l PTHrP-(1–36) increased (2-fold over control) MAPK activation in human platelets. PTH1R was detected in platelets, and the number of platelets expressing it on their surface in patients during AMI (acute myocardial infarction) was not different from...
Clin Sci (Lond) (2007) 112 (1): 43–49.
Published: 01 December 2006
... in patients with AMI (acute myocardial infarction) and, if so, MMP production may be attenuated by statins (3-hydroxy-3-methylglutaryl-CoA reductase inhibitors) through their cardiovascular protective actions. We studied 30 patients, ten control patients with stable angina pectoris and 20 patients with AMI...
Clin Sci (Lond) (2005) 109 (4): 389–395.
Published: 23 September 2005
... and vascular calcification. In the present study, we have investigated serum OPG and RANKL (receptor activator of nuclear factor κB ligand) concentrations in patients with ST elevation AMI (acute myocardial infarction) and established CAD (coronary artery disease). OPG and RANKL were measured in 58 male...
Clin Sci (Lond) (2005) 108 (4): 339–347.
Published: 22 March 2005
...Yudai SHIMODA; Mamoru SATOH; Motoyuki NAKAMURA; Tomonari AKATSU; Katsuhiko HIRAMORI TACE [TNF-α (tumour necrosis factor-α)-converting enzyme] plays an essential role in the shedding of TNF-α, which could affect the outcome of AMI (acute myocardial infarction). To investigate the clinical...
Clin Sci (Lond) (2004) 107 (5): 461–466.
Published: 26 October 2004
...-atherogenic and fibrinolytic effects. However, the association between the 161T allele in exon 6 of the PPARγ gene and premature AMI (acute myocardial infarction) is not clear. We recruited 146 patients with premature AMI (onset age ≤50 years) and 146 controls. The C161T polymorphism was examined using PCR...
Clin Sci (Lond) (2003) 105 (1): 39–44.
Published: 01 July 2003
...Tomonari AKATSU; Motoyuki NAKAMURA; Mamoru SATOH; Katsuhiko HIRAMORI Correspondence: Dr Tomonari Akatsu (e-mail akatsu@carrot.ocn.ne.jp ). 20 12 2002 26 2 2003 26 2 2003 © 2003 The Biochemical Society 2003 acute myocardial infarction leucocyte tumour necrosis...
Clin Sci (Lond) (1999) 96 (2): 129–136.
Published: 01 February 1999
...) increase in an early phase of acute myocardial infarction. However, the relations between plasma BNP levels and left ventricular remodelling, which occurs long after acute myocardial infarction, are not fully understood. Venous plasma BNP levels were measured 2, 7, 14, 30, 90 and 180 days after the onset...
Clin Sci (Lond) (1998) 94 (2): 135–139.
Published: 01 February 1998
... the pathophysiological significance of adrenomedullin in patients with acute myocardial infarction, we measured plasma levels of adrenomedullin. 2. Cardiac catheterization was performed on admission, after 1 day, and after 4 weeks in 36 patients with acute myocardial infarction. We measured plasma levels...
Clin Sci (Lond) (1995) 89 (2): 171–176.
Published: 01 August 1995
... polymorphonuclear neutrophil activation in vitro using plasma samples collected from 14 patients with acute myocardial infarction at time of admission and 6 h and 1, 2, 5 and 7 days later. Plasma samples were incubated with washed polymorphonuclear neutrophils isolated from healthy donors. Expression of adhesion...
Clin Sci (Lond) (1983) 65 (4): 441–444.
Published: 01 October 1983
... not affected in the acute phase of myocardial infarction. 5. Passive head-up tilt (30°) induced vasoconstriction in skeletal muscle tissue. There were no differences in this response at any time during the course of acute myocardial infarction. 6. The abolition of the vasoconstrictor response to increase...
Clin Sci (Lond) (1982) 62 (2): 243–245.
Published: 01 February 1982
...K. Skagen 1. The effect of locally applied subatmospheric pressure on subcutaneous blood flow was studied in six patients with acute myocardial infarction. 2. Blood flow was measured by the local 133 Xe washout technique. 3. Application of subatmospheric pressures of −80 and −150 mmHg...
Clin Sci (Lond) (1981) 60 (2): 213–216.
Published: 01 February 1981
... on day 1 after coronary occlusion but gradually recurred during days 2, 3 and 7. 4. Passive 30° head-up tilt induced a vasoconstriction in subcutaneous tissue. There was no difference in this response at any time during the course of acute myocardial infarction. The vasoconstriction could be blocked...
Clin Sci (Lond) (1981) 60 (2): 157–164.
Published: 01 February 1981
... the vasoconstrictor response appears to be suppressed by a centrally elicited increase in sympathetic discharge rate. acute myocardial infarction autoregulation catecholamines subcutaneous blood flow vasoconstrictor response © 1981 The Biochemical Society and the Medical Research Society 1981 23 7...