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Keywords: non-alcoholic steatohepatitis
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Clin Sci (Lond) (2019) 133 (22): 2245–2264.
Published: 19 November 2019
... and concerted action of factors that contribute to the development of liver inflammation and eventually fibrosis. Here, we summarize fundamental molecular mechanisms underlying the pathogenesis of non-alcoholic steatohepatitis (NASH), how they are interrelated and possible translation to clinical...
Clin Sci (Lond) (2016) 130 (20): 1793–1806.
Published: 09 September 2016
... tissue inflammation, as a novel target of the action of berberine in the liver. acetaminophen toxicity chemokines cytokines LPS macrophages inflammation non-alcoholic steatohepatitis • The use of herbal and dietary supplements for the treatment of liver diseases is growing, but...
Includes: Supplementary data
Clin Sci (Lond) (2014) 126 (4): 297–304.
Published: 22 October 2013
...Gonenc Kocabay; Can Yucel Karabay; Yasar Colak; Vecih Oduncu; Arzu Kalayci; Taylan Akgun; Ahmet Guler; Cevat Kirma The presence of the metabolic syndrome is a strong predictor for the presence of NASH (non-alcoholic steatohepatitis) in patients with NAFLD (non-alcoholic fatty liver disease). In the...
Clin Sci (Lond) (2012) 123 (12): 669–680.
Published: 10 August 2012
...-adrenergic agonist MTX (methoxamine). Besides promoting hepatic fibrosis, the role of hyperleptinaemia in the modulation of vascular response in NASH (non-alcoholic steatohepatitis) rat livers remains unknown. The aim of the present study was to explore the possible links between hyperleptinaemia and the...
Clin Sci (Lond) (2012) 123 (7): 459–471.
Published: 07 June 2012
...) (or monocyte chemoattractant protein-1) regulates inflammatory cell infiltration in the liver and adipose tissue, favouring steatosis. However, its role in the pathogenesis of steatohepatitis is still uncertain. In the present study, we investigated the development of non-alcoholic steatohepatitis...
Clin Sci (Lond) (2006) 111 (2): 135–143.
Published: 13 July 2006
... identifying altered metabolic pathways. The present study was conducted on 39 (20 lean and 19 obese) hypertransaminasemic patients with histologically proven NAFLD {ranging from simple steatosis to severe steatohepatitis [NASH (non-alcoholic steatohepatitis)] and fibrosis} and 28 (20 lean and eight overweight...