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Keywords: transcription factor
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Clin Sci (Lond) (2016) 130 (14): 1165–1177.
Published: 01 June 2016
.... The seven E. coli ribosomal RNA operon upstream regulatory regions differ in structure and transcription factor binding efficiencies Biol. Chem. 2005 386 523 534 10.1515/BC.2005.062 16006239 112 Afflerbach H. Schröder O. Wagner R. Conformational changes of the upstream DNA...
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Clin Sci (Lond) (2014) 127 (3): 171–183.
Published: 03 April 2014
... (nuclear factor κB) is a transcription factor involved in a wide variety of cellular functions, including inflammation and cell survival. Abnormal activation of NF-κB occurs in many pathological conditions [ 2 ]. The transcriptional co-activator p300 is a ubiquitous nuclear phosphoprotein...
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Clin Sci (Lond) (2014) 126 (5): 315–328.
Published: 25 October 2013
... transcription factor © The Authors Journal compilation © 2014 Biochemical Society 2014 28 5 2013 9 8 2013 21 8 2013 Correspondence: Dr Sanna Lehtonen (email [email protected] ). Expression of nephrin has been intensely studied in both human diseases...
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Clin Sci (Lond) (2013) 124 (3): 139–152.
Published: 02 October 2012
... to the inflammation pathways in diabetic nephropathy include transcription factors, pro-inflammatory cytokines, chemokines, adhesion molecules, Toll-like receptors, adipokines and nuclear receptors, which are candidates for the new molecular targets for the treatment of diabetic nephropathy. Understanding...
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Clin Sci (Lond) (2002) 103 (s2002): 16S–20S.
Published: 01 September 2002
.... cardiac hypertrophy endothelin-1 peroxisome proliferator-activated receptor γ transcription factor 16S Clinical Science (2002) 103 (Suppl. 48), 16S 20S (Printed in Great Britain) Peroxisome proliferator-activated receptor-c activators inhibit endothelin-1-related cardiac hypertrophy in rats Satoshi...
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Clin Sci (Lond) (1998) 94 (6): 557–572.
Published: 01 June 1998
... interaction between glucocorticoid receptors and GRE, as these binding sites are absent from the promoter regions of most inflammatory genes. It is more likely to be due to a direct inhibitory interaction between activated glucocorticoid receptors and activated transcription factors, such as nuclear factor-κB...